Muscatine

Aha....so you can smell a difference, even when the winds mix the supposed two together?  What would you say is the predominant bouquet of the turkey farm, as compared to the alleged Muscatine odor?  Is it gamey, or more in the excrement vein?  Does this mean that indeed, all cities have some odors of some kind?

     I caution you, it's possible that Malodery has unusually large and widely separated nostrils.  This just might allow him/her/it to gain some degree of separation of oders wafting on the breeze.  And allow him/her/it to discriminate between the rotting turkey type oders and the more normal and traditional burned popcorn type oder eminating from the alcohol plant here in Muscatine.   In fact he/she/it may have already designated one nostril for one type of oder and the other nostril for another type.  

     I don't think it probable that her/his/its brain is large enough to do the separation and discrimination in the smallish olfactory cortices.

     Now all of the this is speculation.  And as can be seen by the following technical explaination, is probably too complex for an organism such as he/she/it.:

The binding of the ligand (odor molecule or odorant) to the receptor leads to an action potential in the receptor neuron, via a second messenger pathway, depending on the organism. In mammals the odorants stimulate adenylate cyclase to synthesize CAMP via a G protein called G_olf. CAMP, which is the second messenger here, opens a cyclic nucleotide-gated ion channel (CNG) producing an influx of cations (largely Ca²⁺ with some Na⁺) into the cell, slightly depolarising it. The Ca²⁺ in turn opens a Ca²⁺-activated chloride channel, leading to efflux of Cl^-, further depolarising the cell and triggering an action potential. Ca²⁺ is then extruded through a sodium-calcium exchanger. A calcium-calmodulin complex also acts to inhibit the binding of CAMP to the CAMP-dependent channel, thus contributing to olfactory adaptation. This mechanism of transduction is somewhat unique, in that CAMP works by directly binding to the ion channel rather than through activation of protein kinase A. It is similar to the transduction mechanism for photoreceptors, in which the second messenger cGMP works by directly binding to ion channels, suggesting that maybe one of these receptors was evolutionarily adapted into the other. There are also considerable similarities in the immediate processing of stimuli by lateral inhibition. Averaged activity of the receptor neurons can be measured in several ways. In vertebrates responses to an odor can be measured by an electro-olfactogram or through calcium imaging of receptor neuron terminals in the olfactory bulb. In insects, one can perform electroantenogram or also calcium imaging within the olfactory bulb.

The receptor neurons in the nose are particularly interesting because they are the only direct recipient of stimuli in all of the senses which are nerves. Senses like hearing, tasting, and, to some extent, touch use cilia or other indirect pressure to stimulate nerves, and sight uses the chemical rhodopsin to stimulate the brain.

Correction "oder" should be "odor".   I knew what I meant.  Did you?

Soooooo, what you're saying then is that she's full of hooey?